Bettie M Steinberg | |
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Born | 13 June 1937 |
Nationality | American |
Education | University of California, Riverside, Adelphi University, SUNY Stony Brook |
Occupation | Scientist |
Employer | The Feinstein Institute for Medical Research |
Known for | Medical Research |
Website |
Bettie M. Steinberg (born 13 June 1937) is an American scientist who studies molecular biology with a focus on the pathophysiology of human papillomaviruses (HPVs).
Steinberg completed her undergraduate degree at the University of California, Riverside in 1959. She earned a Master's degree in biology from Adelphi University. [1] In 1973, she returned to academia for her PhD in microbiology, which she received from the State University of New York, Stony Brook, in 1976 for her work on bacterial viruses; [2] she then did a post-doctoral fellowship at SUNY Stony Brook, studying mammalian tumor viruses.
After her post-doctoral fellowship, Steinberg moved to Columbia University, where she spent two years as a Senior Research Associate. She then joined the Department of Otolaryngology at Long Island Jewish Medical Center and joined The Feinstein Institute for Medical Research when it was established in 2000. She then served as senior advisor and professor at The Feinstein Institute for Medical Research, [3] provost at the Elmezzi Graduate School of Molecular Medicine (both in Manhasset, New York), and professor at the Department of Molecular Medicine at the Hofstra Northwell School of Medicine in Hempstead, NY. [4]
Steinberg began her research on human papillomaviruses (HPVs) and their association with diseases of the head and neck, particularly recurrent respiratory papillomatosis, at Long Island Jewish Medical Center. These studies were supported by grants from the National Institute of Health (NIH) beginning in 1983. [2]
In collaboration with Dr. Allan Abramson, Steinberg contributed to knowledge that HPVs can establish latent infections and can serve as the source of recurrent disease. [5] Steinberg also contributed to the study of the cell signaling cascades of papilloma cells. [6]