Victor Johnston

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Victor S. Johnston (born 4 May 1943) is an Irish-born psychologist whose work emphasis is emotion, and event related potentials. His areas of study include cognitive engineering, biopsychology, and cognitive psychology. His major research interests are evolutionary psychology, electrophysiology and genetic algorithms. Dr. Johnston states, "The human brain did not evolve to accurately represent the world around us; it evolved only to enhance the survival of our genes." According to Johnston, the combination of emotions with symbolic thought produces meaning. But with this capacity comes the ability to develop meanings for things that do not exist. Little girls develop the ability to attach emotional feelings to dolls, and pretend that their toys live. Little boys learn how to pretend to hunt and fight and attach emotions to them. We learn feelings of desire, fear, and wonder by wandering to the limits of our play. Imagination allows us to create technology, mathematics, and art, but with it can also come terrifying thoughts that could cause harm to us. We grow to learn the difference between most of our thoughts and what they represent, but most of us get fooled into believing the reality of some things that don't exist at all.

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Dr. Johnston received his B.Sc. in psychology, 1964, Queens University, Belfast, N. Ireland; and Ph.D. in psychopharmacology, 1967, University of Edinburgh, Scotland. [1] His doctoral research on the biochemistry of Schizophrenia was awarded the A.E. Bennett Neuropsychiatric Research Foundation Award.

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<span class="mw-page-title-main">Visual N1</span>

The visual N1 is a visual evoked potential, a type of event-related electrical potential (ERP), that is produced in the brain and recorded on the scalp. The N1 is so named to reflect the polarity and typical timing of the component. The "N" indicates that the polarity of the component is negative with respect to an average mastoid reference. The "1" originally indicated that it was the first negative-going component, but it now better indexes the typical peak of this component, which is around 150 to 200 milliseconds post-stimulus. The N1 deflection may be detected at most recording sites, including the occipital, parietal, central, and frontal electrode sites. Although, the visual N1 is widely distributed over the entire scalp, it peaks earlier over frontal than posterior regions of the scalp, suggestive of distinct neural and/or cognitive correlates. The N1 is elicited by visual stimuli, and is part of the visual evoked potential – a series of voltage deflections observed in response to visual onsets, offsets, and changes. Both the right and left hemispheres generate an N1, but the laterality of the N1 depends on whether a stimulus is presented centrally, laterally, or bilaterally. When a stimulus is presented centrally, the N1 is bilateral. When presented laterally, the N1 is larger, earlier, and contralateral to the visual field of the stimulus. When two visual stimuli are presented, one in each visual field, the N1 is bilateral. In the latter case, the N1's asymmetrical skewedness is modulated by attention. Additionally, its amplitude is influenced by selective attention, and thus it has been used to study a variety of attentional processes.

References

  1. Johnston, Victor Samuel (1967). "The effects of mescaline analogues on the operant behaviour of rats".{{cite journal}}: Cite journal requires |journal= (help)