A cognitive vulnerability in cognitive psychology is an erroneous belief, cognitive bias, or pattern of thought that predisposes an individual to psychological problems. [1] The vulnerability exists before the symptoms of a psychological disorder appear. [2] After the individual encounters a stressful experience, the cognitive vulnerability shapes a maladaptive response that increases the likelihood of a psychological disorder. [1]
In psychopathology, there are several perspectives from which the origins of cognitive vulnerabilities can be examined, It is the path way of including cognitive schema models, hopelessness models, and attachment theory. [3] Attentional bias is one mechanism leading to faulty cognitive bias that leads to cognitive vulnerability. Allocating a danger level to a threat depends on the urgency or intensity of the threshold. Anxiety is not associated with selective orientation. [4]
Preliminary or "distal" causes contribute to the formation of a cognitive vulnerability that ultimately, via immediate or proximal causes, leads to the individual manifesting symptoms of the disorder. Immediate cognitive and emotional responses trigger imagery and assumptions formed in the past leading to offsetting, defensive behavior and in turn reinforcing mistaken beliefs or other cognitive vulnerabilities. [1]
The contact made with caretakers determines a certain attachment process. When secure attachment is disrupted and starts to become insecure, abnormal patterns begin, increasing risk for depression. Working models build perceptions of relationships with others. Cognitive vulnerability is created with maladaptive cognitive processing when building relationships and attachments. [3]
Diathesis contributes to vulnerability. [5] The diathesis refers to the inclination to illness. In the diathesis-stress relationship, hidden vulnerability is activated through events that the individual perceives as stressful. Vulnerability in psychological terms is implied as an increased probability of emotional pain and some type of psychopathology. Vulnerability can be a combination and interaction of genetic or acquired experiences. Vulnerability leads to putting up with something unpleasant and represents symptoms of various psychological disorders. Vulnerability predisposes individuals to a disorder, but does not initiate the disorder. Depending on the individual's subjective perception of an event, the diathesis leads to a certain psychological illness. [5]
Through several cognitive biases, selective mood-congruent cues become established over long intervals. Emotional stimuli matching the emotional concerns create an aggregate effect on symptoms related to depression. Depression is associated with selective orientation. It prevents attention toward emotional cues that do not fit the internalized scheme to which the individual has become vulnerable, and leads to comorbid anxiety. When individuals who are prone to depression are asked to recall a specific event, they explain the general class of events (e.g., "The time when I was living with my parents"). [4]
Associative and reflective processing mechanisms apply when cognitive vulnerability is processed into depression. The dual process model is valid in social and personality psychology but is not adapted to clinical phenomena. Negative bias in self-assessment provides a foundation for a cognitive vulnerability to depression. Then a downward spiral forms to create forms of dysphoria. Negatively biased associative processing will maintain a dysphoric mood state. As the dysphoric mood escalates, cognitive resources necessary to combating dysphoria by reflective processing are depleted. Irrelevant tasks and intrusive thoughts come to mind when in a dysphoric mood, and cognitive resource depletion further contributes to mood escalation. [6]
The feedback loop in the dual process model is between self referent cognition and dysphoria. The feedback loop establishes an inability to apply reflective processing to correct negative biases. [6]
Postponing the reflective processes leads to mood persistence. The individual becomes accustomed to a state of dysphoria as they experience more and more negative mood states. Dysphoric moods create more associative processing for depressive vulnerable people by negative cognitive biases. When associative bias gets stronger, the bias becomes difficult to override. Ineffective reflective strategies lead to persistence of dysphoric moods. [6]
The likelihood of another depressive episode escalates with the number of previous episodes. A depressive episode by itself is a vulnerability factor. Each episode of depression makes it easier for the neurotransmitter system to become deregulated. A strong stressor is needed for the initializing first episode; however, subsequent episodes can be triggered by increasingly mild stressors. Contextual information develops such that small changes in mood are sufficient to activate vulnerability. Weakening and frequency of depressive episodes triggers the biological processes related to the initial episode. Depressive episodes are experienced as having no control over traumatic events. A depressive condition results in social rejection and lowered self esteem, leading to further depressive symptoms. [5]
Schemas in depression are formed in association with stressful events in childhood and condition the individual to respond in an abnormal manner to life experiences that recall those childhood traumas. [3] During childhood and adolescence, the individual who is prone to depression begins to match life situations with prototypes of specific stressful experiences from childhood. The cognitive vulnerability thus manifests itself. [3]
Negative events during childhood lead the child to internalize negative events. Just as repeated positive experiences lead the child to develop a positive self image and optimism regarding future events, negative events lead to the development of expectations of hopelessness or even depression when the individual faces a stressful situation in the future. [3]
A study of people with bipolar disorder found that, compared with non-bipolar controls, they had significantly higher levels of dysfunctional attitudes such as perfectionism and need for approval that increase their cognitive vulnerability to depression. [7]
A mental disorder is an impairment of the mind disrupting normal thinking, feeling, mood, behavior, or social interactions, and accompanied by significant distress or dysfunction. The causes of mental disorders are very complex and vary depending on the particular disorder and the individual. Although the causes of most mental disorders are not fully understood, researchers have identified a variety of biological, psychological, and environmental factors that can contribute to the development or progression of mental disorders. Most mental disorders result in a combination of several different factors rather than just a single factor.
Psychology is an academic and applied discipline involving the scientific study of human mental functions and behavior. Occasionally, in addition or opposition to employing the scientific method, it also relies on symbolic interpretation and critical analysis, although these traditions have tended to be less pronounced than in other social sciences, such as sociology. Psychologists study phenomena such as perception, cognition, emotion, personality, behavior, and interpersonal relationships. Some, especially depth psychologists, also study the unconscious mind.
A mood swing is an extreme or sudden change of mood. Such changes can play a positive part in promoting problem solving and in producing flexible forward planning, or be disruptive. When mood swings are severe, they may be categorized as part of a mental illness, such as bipolar disorder, where erratic and disruptive mood swings are a defining feature.
Depression is a mental state of low mood and aversion to activity. It affects more than 280 million people of all ages. Depression affects a person's thoughts, behavior, feelings, and sense of well-being. Depressed people often experience loss of motivation or interest in, or reduced pleasure or joy from, experiences that would normally bring them pleasure or joy. Depressed mood is a symptom of some mood disorders such as major depressive disorder and dysthymia; it is a normal temporary reaction to life events, such as the loss of a loved one; and it is also a symptom of some physical diseases and a side effect of some drugs and medical treatments. It may feature sadness, difficulty in thinking and concentration and a significant increase or decrease in appetite and time spent sleeping. People experiencing depression may have feelings of dejection or hopelessness and may experience suicidal thoughts. It can either be short term or long term.
Links between creativity and mental health have been extensively discussed and studied by psychologists and other researchers for centuries. Parallels can be drawn to connect creativity to major mental disorders including bipolar disorder, schizophrenia, major depressive disorder, anxiety disorder, OCD and ADHD. For example, studies have demonstrated correlations between creative occupations and people living with mental illness. There are cases that support the idea that mental illness can aid in creativity, but it is also generally agreed that mental illness does not have to be present for creativity to exist.
Dysphoria is a profound state of unease or dissatisfaction. It is the semantic opposite of euphoria. In a psychiatric context, dysphoria may accompany depression, anxiety, or agitation.
Depressive realism is the hypothesis developed by Lauren Alloy and Lyn Yvonne Abramson that depressed individuals make more realistic inferences than non-depressed individuals. Although depressed individuals are thought to have a negative cognitive bias that results in recurrent, negative automatic thoughts, maladaptive behaviors, and dysfunctional world beliefs, depressive realism argues not only that this negativity may reflect a more accurate appraisal of the world but also that non-depressed individuals' appraisals are positively biased.
The diathesis-stress model, also known as the vulnerability–stress model, is a psychological theory that attempts to explain a disorder, or its trajectory, as the result of an interaction between a predispositional vulnerability, the diathesis, and stress caused by life experiences. The term diathesis derives from the Greek term (διάθεσις) for a predisposition or sensibility. A diathesis can take the form of genetic, psychological, biological, or situational factors. A large range of differences exists among individuals' vulnerabilities to the development of a disorder.
Mindfulness-based cognitive therapy (MBCT) is an approach to psychotherapy that uses cognitive behavioral therapy (CBT) methods in conjunction with mindfulness meditative practices and similar psychological strategies. The origins to its conception and creation can be traced back to the traditional approaches from East Asian formative and functional medicine, philosophy and spirituality, birthed from the basic underlying tenets from classical Taoist, Buddhist and Traditional Chinese medical texts, doctrine and teachings.
Bipolar II disorder (BP-II) is a mood disorder on the bipolar spectrum, characterized by at least one episode of hypomania and at least one episode of major depression. Diagnosis for BP-II requires that the individual must never have experienced a full manic episode. Otherwise, one manic episode meets the criteria for bipolar I disorder (BP-I).
Behavioral theories of depression explain the etiology of depression based on the behavioural sciences, and they form the basis for behavioral therapies for depression.
Rumination is the focused attention on the symptoms of one's mental distress, and on its possible causes and consequences, as opposed to its solutions, according to the Response Styles Theory proposed by Nolen-Hoeksema in 1998.
Meta-mood is a term used by psychologists to refer to an individual's awareness of their emotions. The term was first utilized by John D. Mayer and Peter Salovey who believed the experience of mood involved "direct" and "indirect" components. While the direct level refers to the simple appearance of mood - happiness, fear, anger, sadness, and surprise, the indirect level, or the meta-mood experience, does not solely consist of the emotions experienced by an individual in the moment. Rather, it is a reflective state which involves additional thoughts and feelings about the mood itself. "I shouldn’t feel this way" or "I am thinking of ways to improve my mood" are examples of reflective thoughts during a meta-mood experience.
The associated features of bipolar disorder are clinical phenomena that often accompany bipolar disorder (BD) but are not part of the diagnostic criteria for the disorder. There are several childhood precursors in children who later receive a diagnosis of bipolar disorder. They may show subtle early traits such as mood abnormalities, full major depressive episodes, and attention-deficit hyperactivity disorder. BD is also accompanied by changes in cognition processes and abilities. This includes reduced attentional and executive capabilities and impaired memory. How the individual processes the world also depends on the phase of the disorder, with differential characteristics between the manic, hypomanic and depressive states. Some studies have found a significant association between bipolar disorder and creativity.
Overgeneral autobiographical memory (OGM) is an inability to retrieve specific memories from one's autobiographical memory. Instead, general memories are recalled, such as repeated events or events occurring over broad periods. For example, when asked to recall a happy event, a person who exhibits OGM may say, "when I was on vacation last month" instead of remembering a single incident, such as, "my high school graduation." Research shows a correlation between OGM and certain mental illnesses, such as major depressive disorder (MDD) and posttraumatic stress disorder (PTSD).
Self-blame is a cognitive process in which an individual attributes the occurrence of a stressful event to oneself. The direction of blame often has implications for individuals’ emotions and behaviors during and following stressful situations. Self-blame is a common reaction to stressful events and has certain effects on how individuals adapt. Types of self-blame are hypothesized to contribute to depression, and self-blame is a component of self-directed emotions like guilt and self-disgust. Because of self-blame's commonality in response to stress and its role in emotion, self-blame should be examined using psychology's perspectives on stress and coping. This article will attempt to give an overview of the contemporary study on self-blame in psychology.
The following outline is provided as an overview of and topical guide to bipolar disorder:
The Vulnerability-Stress-Adaptation (VSA) Model is a framework for conceptualizing the dynamic processes of marriage, created by Benjamin Karney and Thomas Bradbury. The VSA Model emphasizes the consideration of multiple dimensions of functioning, including couple members’ enduring vulnerabilities, experiences of stressful events, and adaptive processes, to account for variations in marital quality and stability over time. The VSA model was a departure from past research considering any one of these themes separately as a contributor to marital outcomes, and integrated these separate factors into a single, cohesive framework in order to best explain how and why marriages change over time. In adherence with the VSA model, in order to achieve a complete understanding of marital phenomenon, research must consider all dimensions of marital functioning, including enduring vulnerabilities, stress, and adaptive processes simultaneously.
Emotions play a key role in overall mental health, and sleep plays a crucial role in maintaining the optimal homeostasis of emotional functioning. Deficient sleep, both in the form of sleep deprivation and restriction, adversely impacts emotion generation, emotion regulation, and emotional expression.
In the context of the nature-nurture debate, interactionism is the view that all human behavioral traits develop from the interaction of both "nature" and "nurture", that is, from both genetic and environmental factors. This view further holds that genetic and environmental influences on organismal development are so closely interdependent that they are inseparable from one another. Historically, it has often been confused with the statistical concept of gene-environment interaction. Historically, interactionism has presented a limited view of the manner in which behavioral traits develop, and has simply demonstrated that "nature" and "nurture" are both necessary. Among the first biologists to propose an interactionist theory of development was Daniel Lehrman. Since then, numerous interactionist perspectives have been proposed, and the contradictions between many of these perspectives has led to much controversy in evolutionary psychology and behavioral genetics. Proponents of various forms of interactionist perspectives include Philip Kitcher, who refers to his view as "causal democracy", and Susan Oyama, who describes her perspective as "constructive interactionism". Critics of interactionism include major figures in behavioral genetics such as Arthur Jensen, Robert Plomin, and philosopher Neven Sesardic.