List of cardiology mnemonics

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This is a list of cardiology mnemonics , categorized and alphabetized. For mnemonics in other medical specialities, see this list of medical mnemonics.

Contents

Aortic regurgitation: causes

CREAM:

Congenital

Rheumatic damage

Endocarditis

Aortic dissection/ Aortic root dilatation

Marfan’s

Aortic stenosis characteristics

SAD: [1] p. 29

Syncope

Angina

Dyspnoea

Aortic to right Subclavian path

ABC'S [1] p. 1

Aortic arch gives rise to:

Brachiocephalic trunk

Left Common Carotid

Left Subclavian

Heart valves (right to left)

Toilet Paper My Ass, or They Pay Me Alcohol, or "T"hugs "P"ush "Me" "A"round. [2]

Tricuspid valve

Pulmonary semilunar valve

Mitral (bicuspid) valve

Aortic semilunar valve

Apex beat: abnormalities found on palpation, causes of impalpable

HILT: [1] p. 29

Heaving

Impalpable

Laterally displaced

Thrusting/ Tapping

If it's impalpable, causes are COPD: [1] p. 29

COPD

Obesity

Pleural, Pericardial effusion

Dextrocardia

Atrial Arrhythmias

Anticoagulants: To prevent embolization.

Beta blockers: To block the effects of certain hormones on the heart to slow the heart rate.

Calcium Channel Blockers: Help slow the heart rate by blocking the number of electrical impulses that pass through the AV node into the lower heart chambers (ventricles).

Digoxin: Helps slow the heart rate by blocking the number of electrical impulses that pass through the AV node into the lower heart chambers (ventricles).

Electrocardioversion: A procedure in which electric currents are used to reset the heart's rhythm back to regular pattern. [3]

Atrial Fibrillation causes

Pirates: [1] p. 3

Pulmonary: PE, COPD

Iatrogenic

Rheumatic heart: mitral regurgitation

Atherosclerotic: MI, CAD

Thyroid: hyperthyroid

Endocarditis

Sick sinus syndrome

Atrial fibrillation management

ABCD: [1] p. 30

Anti-coagulate

Beta-block to control rate

Cardiovert

Digoxin

Beck's triad (cardiac tamponade)

3 D's: [1] p. 30

Diminished heart sounds

Distended jugular veins

Decreased arterial pressure

Betablockers: cardioselective betablockers

Betablockers Acting Exclusively At Myocardium: [1] p. 30

Betaxolol

Acebutelol

Esmolol

Atenolol

Metoprolol

CHF Treatment

LMNOP

Lasix

Morphine

Nitrites

Oxygen

VassoPressors [4]

CHF: causes of exacerbation

FAILURE [1] p. 30

Forgot medication

Arrhythmia/ Anaemia

Ischemia/ Infarction/ Infection

Lifestyle: taken too much salt

Upregulation of CO: pregnancy, hyperthyroidism

Renal failure

Embolism: pulmonary

Complications of Myocardial Infarction

Darth Vader

Death

Arrhythmia

Rupture(free ventricular wall/ ventricular septum/ papillary muscles)

Tamponade

Heart failure (acute or chronic)

Valve disease

Aneurysm of Ventricles

Dressler's Syndrome

thromboEmbolism (mural thrombus)

Recurrence/ mitral Regurgitation [5]

Coronary artery bypass graft: indications

DUST: [1] p. 31

Depressed ventricular function

Unstable angina

Stenosis of the left main stem

Triple vessel disease

ECG: left vs. right bundle block

WiLLiaMMaRRoW: [1] p. 31

W pattern in V1-V2 and M pattern in V3-V6 is Left bundle block.

M pattern in V1-V2 and W in V3-V6 is Right bundle block.

Exercise ramp ECG: contraindications

RAMP: [1] p. 31

Recent MI

Aortic stenosis

MI in the last 7 days

Pulmonary hypertension

Endocarditis

FROM JANE:

Fever

Roth's spots

Osler's nodes

Murmur of heart

Janeway lesions

Anemia

Emboli

Heart valve sequence

TryPuling MyAorta: [1] p. 3

Tricuspid

Pulmonary

Mitral (bicuspid)

Aorta

Heart blocks

If the R is far from P, then you have a First Degree.

Longer, longer, longer, drop! Then you have a Wenkebach.

if some P's don't get through, then you have Mobitz II.

If P's and Q's don't agree, then you have a Third Degree. [6]

Infarctions

INFARCTIONS [1] p. 34

IV access

Narcotic analgesics (e.g. morphine, pethidine)

Facilities for defibrillation (DF)

Aspirin/ Anticoagulant (heparin)

Rest

Converting enzyme inhibitor

Thrombolysis

IV beta blocker

Oxygen 60%

Nitrates

Stool Softeners

JVP: wave form

ASK ME [1] p. 32

Atrial contraction

Systole (ventricular contraction)

Klosure (closure) of tricuspid valve, so atrial filling

Maximal atrial filling

Emptying of atrium

MI: basic management

BOOMAR: [1] p. 32

Bed rest

Oxygen

Opiate

Monitor

Anticoagulate

Reduce clot size

MI: signs and symptoms

PULSE: [1] p. 32

Persistent chest pains

Upset stomach

Lightheadedness

Shortness of breath

Excessive sweating

MI: therapeutic treatment

O BATMAN! [1] p. 32

Oxygen

Beta blocker

ASA

Thrombolytics (e.g. heparin)

Morphine

Ace prn

Nitroglycerin

MI: treatment of acute MI

COAG: [1] p. 32

Cyclomorph

Oxygen

Aspirin

Glycerol trinitrate

Murmur attributes

"IL PQRST" (person has ill PQRST heart waves): [1] p. 32

Intensity

Loccasion

Pitch

Quality

Radiation

Shape

Timing

Murmurs: innocent murmur features

8 S's: [1] p. 32

Soft

Systolic

Short

Sounds (S1 & S2) normal

Symptomless

Special tests normal (X-ray, EKG)

Standing/ Sitting (vary with position)

Sternal depression

Murmurs: louder with inspiration vs expiration

LEft sided murmurs louder with Expiration

RIght sided murmurs louder with Inspiration. [1] p. 32

Murmurs: questions to ask

SCRIPT: [1] p. 32

Site

Character (e.g. harsh, soft, blowing)

Radiation

Intensity

Pitch

Timing

Murmurs: systolic vs. diastolic

PASS:Pulmonic & Aortic

Stenosis=Systolic.

PAID: Pulmonic & Aortic

Insufficiency=Diastolic. [1] p. 32

Pericarditis: causes

CARDIAC RIND: [1] p. 34

Collagen vascular disease

Aortic aneurysm

Radiation

Drugs (such as hydralazine)

Infections

Acute renal failure

Cardiac infarction

Rheumatic fever

Injury

Neoplasms

Dressler's syndrome

Pericarditis: EKG

PericarditiS: [1] p. 34

PR depression in precordial leads.

ST elevation.

Peripheral vascular insufficiency: inspection criteria

SICVD: [1] p. 34

Symmetry of leg musculature

Integrity of skin

Color of toenails

Varicose veins

Distribution of hair

Pulseless electrical activity: causes

PATCH MED: [1] p. 34

Pulmonary embolus

Acidosis

Tension pneumothorax

Cardiac tamponade

Hypokalemia/ Hyperkalemia/ Hypoxia/ Hypothermia/ Hypovolemia

Myocardial infarction

Electrolyte derangements

Drugs

ST elevation causes in ECG

ELEVATION: [1] p. 34

Electrolytes

LBBB

Early repolarization

Ventricular hypertrophy

Aneurysm

Treatment (e.g. pericardiocentesis)

Injury (AMI, contusion)

Osborne waves (hypothermia)

Non-occlusive vasospasm

Supraventricular tachycardia: treatment

ABCDE: [1] p. 35

Adenosine

Beta-blocker

Calcium channel antagonist

Digoxin

Excitation (vagal stimulation)

Ventricular tachycardia: treatment

LAMB: [1] p. 35

Lidocaine

Amiodarone

Mexiltene/ Magnesium

Beta-blocker

White Blood Cell Count

Never let monkeys eat bananas:

Neutrophils

lymphocytes

monocytes

eosinophils

basophils [7]

Related Research Articles

<span class="mw-page-title-main">Heart valve</span> A flap of tissue that prevent backflow of blood around the heart

A heart valve is a biological one-way valve that allows blood to flow in one direction through the chambers of the heart. Four valves are usually present in a mammalian heart and together they determine the pathway of blood flow through the heart. A heart valve opens or closes according to differential blood pressure on each side.

<span class="mw-page-title-main">Heart sounds</span> Noise generated by the beating heart

Heart sounds are the noises generated by the beating heart and the resultant flow of blood through it. Specifically, the sounds reflect the turbulence created when the heart valves snap shut. In cardiac auscultation, an examiner may use a stethoscope to listen for these unique and distinct sounds that provide important auditory data regarding the condition of the heart.

<span class="mw-page-title-main">Mitral valve</span> Valve in the heart connecting the left atrium and left ventricle

The mitral valve, also known as the bicuspid valve or left atrioventricular valve, is one of the four heart valves. It has two cusps or flaps and lies between the left atrium and the left ventricle of the heart. The heart valves are all one-way valves allowing blood flow in just one direction. The mitral valve and the tricuspid valve are known as the atrioventricular valves because they lie between the atria and the ventricles.

<span class="mw-page-title-main">Heart murmur</span> Medical condition

Heart murmurs are unique heart sounds produced when blood flows across a heart valve or blood vessel. This occurs when turbulent blood flow creates a sound loud enough to hear with a stethoscope. The sound differs from normal heart sounds by their characteristics. For example, heart murmurs may have a distinct pitch, duration and timing. The major way health care providers examine the heart on physical exam is heart auscultation; another clinical technique is palpation, which can detect by touch when such turbulence causes the vibrations called cardiac thrill. A murmur is a sign found during the cardiac exam. Murmurs are of various types and are important in the detection of cardiac and valvular pathologies.

<span class="mw-page-title-main">Systole</span> Part of the cardiac cycle when a heart chamber contracts

Systole is the part of the cardiac cycle during which some chambers of the heart contract after refilling with blood. Its contrasting phase is diastole, the relaxed phase of the cardiac cycle when the chambers of the heart are refilling with blood.

<span class="mw-page-title-main">Mitral stenosis</span> Heart disease with narrowing of valve

Mitral stenosis is a valvular heart disease characterized by the narrowing of the opening of the mitral valve of the heart. It is almost always caused by rheumatic valvular heart disease. Normally, the mitral valve is about 5 cm2 during diastole. Any decrease in area below 2 cm2 causes mitral stenosis. Early diagnosis of mitral stenosis in pregnancy is very important as the heart cannot tolerate increased cardiac output demand as in the case of exercise and pregnancy. Atrial fibrillation is a common complication of resulting left atrial enlargement, which can lead to systemic thromboembolic complications such as stroke.

<span class="mw-page-title-main">Ventricular septal defect</span> Medical condition

A ventricular septal defect (VSD) is a defect in the ventricular septum, the wall dividing the left and right ventricles of the heart. The extent of the opening may vary from pin size to complete absence of the ventricular septum, creating one common ventricle. The ventricular septum consists of an inferior muscular and superior membranous portion and is extensively innervated with conducting cardiomyocytes.

<span class="mw-page-title-main">Mitral regurgitation</span> Form of valvular heart disease

Mitral regurgitation (MR), also known as mitral insufficiency or mitral incompetence, is a form of valvular heart disease in which the mitral valve is insufficient and does not close properly when the heart pumps out blood. It is the abnormal leaking of blood backwards – regurgitation from the left ventricle, through the mitral valve, into the left atrium, when the left ventricle contracts. Mitral regurgitation is the most common form of valvular heart disease.

A transthoracic echocardiogram (TTE) is the most common type of echocardiogram, which is a still or moving image of the internal parts of the heart using ultrasound. In this case, the probe is placed on the chest or abdomen of the subject to get various views of the heart. It is used as a non-invasive assessment of the overall health of the heart, including a patient's heart valves and degree of heart muscle contraction. The images are displayed on a monitor for real-time viewing and then recorded.

<span class="mw-page-title-main">Cardiac cycle</span> Performance of the human heart

The cardiac cycle is the performance of the human heart from the beginning of one heartbeat to the beginning of the next. It consists of two periods: one during which the heart muscle relaxes and refills with blood, called diastole, following a period of robust contraction and pumping of blood, called systole. After emptying, the heart relaxes and expands to receive another influx of blood returning from the lungs and other systems of the body, before again contracting to pump blood to the lungs and those systems.

<span class="mw-page-title-main">Valvular heart disease</span> Disease in the valves of the heart

Valvular heart disease is any cardiovascular disease process involving one or more of the four valves of the heart. These conditions occur largely as a consequence of aging, but may also be the result of congenital (inborn) abnormalities or specific disease or physiologic processes including rheumatic heart disease and pregnancy.

<span class="mw-page-title-main">Right ventricular hypertrophy</span> Medical condition

Right ventricular hypertrophy (RVH) is a condition defined by an abnormal enlargement of the cardiac muscle surrounding the right ventricle. The right ventricle is one of the four chambers of the heart. It is located towards the right lower chamber of the heart and it receives Deoxygenated blood from the right upper chamber and pumps blood into the lungs.

Regurgitation is blood flow in the opposite direction from normal, as the backward flowing of blood into the heart or between heart chambers. It is the circulatory equivalent of backflow in engineered systems. It is sometimes called reflux.

<span class="mw-page-title-main">Tricuspid regurgitation</span> Type of valvular heart disease

Tricuspid regurgitation (TR), also called tricuspid insufficiency, is a type of valvular heart disease in which the tricuspid valve of the heart, located between the right atrium and right ventricle, does not close completely when the right ventricle contracts (systole). TR allows the blood to flow backwards from the right ventricle to the right atrium, which increases the volume and pressure of the blood both in the right atrium and the right ventricle, which may increase central venous volume and pressure if the backward flow is sufficiently severe.

<span class="mw-page-title-main">Lutembacher's syndrome</span> Medical condition

Lutembacher's syndrome is a very rare form of congenital heart disease that affects one of the chambers of the heart as well as a valve. It is commonly known as both congenital atrial septal defect (ASD) and acquired mitral stenosis (MS). Congenital atrial septal defect refers to a hole being in the septum or wall that separates the two atria; this condition is usually seen in fetuses and infants. Mitral stenosis refers to mitral valve leaflets sticking to each other making the opening for blood to pass from the atrium to the ventricles very small. With the valve being so small, blood has difficulty passing from the left atrium into the left ventricle. Septal defects that may occur with Lutembacher's syndrome include: Ostium primum atrial septal defect or ostium secundum which is more prevalent.

The following outline is provided as an overview of and topical guide to cardiology, the branch of medicine dealing with disorders of the human heart. The field includes medical diagnosis and treatment of congenital heart defects, coronary artery disease, heart failure, valvular heart disease and electrophysiology. Physicians who specialize in cardiology are called cardiologists.

Cardiac physiology or heart function is the study of healthy, unimpaired function of the heart: involving blood flow; myocardium structure; the electrical conduction system of the heart; the cardiac cycle and cardiac output and how these interact and depend on one another.

References

  1. 1 2 3 4 5 6 7 8 9 10 11 12 13 14 15 16 17 18 19 20 21 22 23 24 25 26 27 28 29 30 31 "Anatomy" (PDF). medicalmnemonics.com. Retrieved 14 February 2015.
  2. Mega List of Mnemonics for Nurses & Nursing Students. Examville Study Guides. 2010.
  3. Pharmacology nursing mnemonics
  4. Myers, Jeffrey W.; Neighbors, Marianne; Tannehill-Jones, Ruth (2002). Principles of Pathophysiology and Emergency Medical Care. Cengage Learning. p. 144. ISBN   9780766825482.
  5. "Cardiovascular Mnemonics - Oxford Medical Education" . Retrieved 2015-08-23.
  6. "The Heart Block Poem". Nurseslabs. tumblr.com.
  7. Connie Allen; Valerie Harper (5 January 2011). Laboratory Manual for Anatomy and Physiology. John Wiley & Sons. pp. 418–. ISBN   978-0-470-59890-0.