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Dens evaginatus | |
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Other names | Tuberculated cusp, accessory tubercle, occlusal tuberculated premolar, Leong's premolar, evaginatus odontoma, occlusal pearl [1] [2] |
Specialty | Dentistry |
Dens evaginatus is a rare odontogenic developmental anomaly that is found in teeth where the outer surface appears to form an extra bump or cusp.
Premolars are more likely to be affected than any other tooth. [3] It could occur unilaterally or bilaterally. [1] Dens evaginatus (DE) typically occurs bilaterally and symmetrically. [4] This may be seen more frequently in Asians [3] (including Chinese, Malay, Thai, Japanese, Filipino and Indian populations). [4]
The prevalence of DE ranges from 0.06% to 7.7% depending on the race. [3] It is more common in men than in women, [3] more frequent in the mandibular teeth than the maxillary teeth. [1] Patients with Ellis-van Creveld syndrome, incontinentia pigmenti achromians, Mohr syndrome, Rubinstein-Taybi syndrome and Sturge Weber syndrome are at a higher risk of having DE. [3] [2]
It is important to diagnose DE early and provide appropriate treatment to help prevent periodontal disease, caries, pulpal complications [3] and malocclusion. [2] It occurs on the cingulum/occlusal surface of the teeth. The extra cusp can cause occlusal interference, displace of the affected tooth and/or opposing teeth, irritates the tongue when speaking and eating and decay the developmental grooves. [2] Temporomandibular joint pain could be experienced secondarily due to occlusal trauma caused by the tubercle. [1] [2]
This cusp could be worn away or fractured easily. [1] [4] [2] In 70% [4] of the cases, the fine pulpal extension were exposed which can lead to infection, [4] pulpal necrosis and periapical pathosis.
The cause of DE is still unclear. [2] There is literature indicating that DE is an isolated anomaly. During the bell stage of tooth formation, DE may occur as a result of an unusual growth and folding of the inner enamel epithelium and ectomesenchymal cells of dental papilla into the stellate reticulum of the enamel organ. [5] [4]
Diagnosis of DE can be difficult when there is no signs and symptoms of necrotic or infected pulp. [1] It is a challenging task to differentiate between a true periapical lesion and a normal periapical radiolucency of a dental follicle of an immature apex. [1]
The anterior DE tubercles have an average width of 3.5mm and length of 6.0mm, [4] while posterior tubercles have an average 2.0mm in width and length of up to 3.5mm. [4] If the cusp of Carabelli is present, the tooth associated are often larger mesiodistally and it is not uncommon that a DE involved tooth has an abnormal root pattern. [4]
There are 4 different ways to classify/ categorize DE involved teeth.
If the tooth involved is asymptomatic or small, no treatment is needed [3] and a preventative approach should be taken.
Preventative measures [3] include:
For teeth with normal pulp and mature apex, reduce the opposing occluding tooth. [4] Reinforce the tubercle by applying flowable composite. [4] [2] Occlusion, restoration, pulp and periapex assessment should be done yearly. [4] When there is adequate pulp recession, tubercle can be removed and tooth can be restored. [4]
For teeth with normal pulp and immature apex, reduce the opposing occluding tooth. [4] Apply flowable composite to the tubercle. [4] Occlusion, restoration, pulp and periapex assessment should be done every 3–4 months until the apex matures. [4] When there is signs of adequate pulp recession, tubercle can be removed and tooth can be restored. [4]
For teeth with inflamed pulp and mature apex, conventional root canal treatment could be carried out and restored accordingly. [4]
For teeth with inflamed pulp and immature apex, shallow MTA pulpotomy could be performed and then restore with glass ionomer and composite. [4]
For teeth with necrotic pulp and mature apex, conventional root canal therapy could be done and restored. [4]
For teeth with necrotic pulp and immature apex, MTA root-end barrier could be carried out. Glass ionomer layer and composite could be used to restore the tooth. [4]
If there is occlusal interference, the opposing projection should be reduced. [3] [2] Make sure that the tubercle does not contact other teeth in all excursive movement. [2] This is usually done over a few appointments, 6 to 8 weeks apart to allow the formation of reparative dentin to protect the pulp. [3] Fluoride varnish should be applied onto the ground surface. [7] [6] [3] [4] Recall the patient for follow-up after 3, 6 and 12 months. [3]
In some cases, extraction [ citation needed ] could be considered (e.g. for orthodontic purposes, failed apexification) [2]
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Toothache, also known as dental pain, is pain in the teeth or their supporting structures, caused by dental diseases or pain referred to the teeth by non-dental diseases. When severe it may impact sleep, eating, and other daily activities.
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The mandibular first molar or six-year molar is the tooth located distally from both the mandibular second premolars of the mouth but mesial from both mandibular second molars. It is located on the mandibular (lower) arch of the mouth, and generally opposes the maxillary (upper) first molars and the maxillary 2nd premolar in normal class I occlusion. The function of this molar is similar to that of all molars in regard to grinding being the principal action during mastication, commonly known as chewing. There are usually five well-developed cusps on mandibular first molars: two on the buccal, two lingual, and one distal. The shape of the developmental and supplementary grooves, on the occlusal surface, are describes as being 'M' shaped. There are great differences between the deciduous (baby) mandibular molars and those of the permanent mandibular molars, even though their function are similar. The permanent mandibular molars are not considered to have any teeth that precede it. Despite being named molars, the deciduous molars are followed by permanent premolars.
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Dens invaginatus (DI), also known as tooth within a tooth, is a rare dental malformation found in teeth where there is an infolding of enamel into dentine. The prevalence of condition is 0.3 - 10%, affecting more males than females. The condition is presented in two forms, coronal and radicular, with the coronal form being more common.
Dentin dysplasia (DD) is a rare genetic developmental disorder affecting dentine production of the teeth, commonly exhibiting an autosomal dominant inheritance that causes malformation of the root. It affects both primary and permanent dentitions in approximately 1 in every 100,000 patients. It is characterized by presence of normal enamel but atypical dentin with abnormal pulpal morphology. Witkop in 1972 classified DD into two types which are Type I (DD-1) is the radicular type, and type II (DD-2) is the coronal type. DD-1 has been further divided into 4 different subtypes (DD-1a,1b,1c,1d) based on the radiographic features.
Talon Cusp is a rare dental anomaly. Generally a person with this develops "cusp-like" projections located on the inside surface of the affected tooth. Talon cusp is an extra cusp on an anterior tooth. Although talon cusp may not appear serious, it can cause clinical, diagnostic, functional problems and alters the aesthetic appeal.
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Root canal treatment is a treatment sequence for the infected pulp of a tooth which is intended to result in the elimination of infection and the protection of the decontaminated tooth from future microbial invasion. Root canals, and their associated pulp chamber, are the physical hollows within a tooth that are naturally inhabited by nerve tissue, blood vessels and other cellular entities. Together, these items constitute the dental pulp.
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In anatomy the apical foramen is the opening at the apex of the root of a tooth, through which the nerve and blood vessels that supply the dental pulp pass. Thus it represents the junction of the pulp and the periodontal tissue.
Pulp necrosis is a clinical diagnostic category indicating the death of cells and tissues in the pulp chamber of a tooth with or without bacterial invasion. It is often the end result of many cases of dental trauma, caries and irreversible pulpitis.
A phoenix abscess is an acute exacerbation of a chronic periapical lesion. It is a dental abscess that can occur immediately following root canal treatment. Another cause is due to untreated necrotic pulp. It is also the result of inadequate debridement during the endodontic procedure. Risk of occurrence of a phoenix abscess is minimised by correct identification and instrumentation of the entire root canal, ensuring no missed anatomy.
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Apexification is a method of dental treatment to induce a calcific barrier in a root with incomplete formation or open apex of a tooth with necrotic pulp. Pulpal involvement usually occurs as a consequence of trauma or caries involvement of young or immature permanent teeth. As a sequelae of untreated pulp involvement, loss of pulp vitality or necrotic pulp took place for the involved teeth.
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