Developmental theory of crime

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In 1993, American psychologist Terrie Moffitt described a dual taxonomy of offending behavior in an attempt to explain the developmental processes that lead to the distinctive shape of the age crime curve. [1] [2] Moffitt proposed that there are two main types of antisocial offenders in society: The adolescence-limited offenders, who exhibit antisocial behavior only during adolescence, and the life-course-persistent offenders, who begin to behave antisocially early in childhood and continue this behavior into adulthood. [3] This theory is used with respect to antisocial behavior instead of crime due to the differing definitions of 'crime' among cultures. Due to similar characteristics and trajectories, this theory can be applied to both females and males. [4]

Contents

Antisocial Personality Disorder

Antisocial personality disorder (ASPD) is recognized by the Diagnostic and Statistical Manual of Mental Disorders (DSM-IV). It is a disorder characterized by a severe disregard for the rights of others. In most of the studies described below, individuals who exhibit antisocial behavior, but have not been diagnosed with ASPD, are used as subjects.

Age and antisocial personality disorder

The number of arrests spikes in adolescence, but subsequently declines. This spike leads people to wonder whether more offenders are appearing or more offenses are committed by the same few offenders. Evidence shows that there is an increase in both. The most persistent 5% of offenders are responsible for more than 50% of known crimes committed. [5]

Several experiments have been conducted to investigate the relationship between extremity and stability of offenses. In one such experiment a group of third grade boys was studied. Out of the most aggressive 5%, 39% of them scored above the 95th percentile on aggression ten years later, and 100% of them were above the median. [5]

Aggression and antisocial behavior in a child is a predictor of adult antisocial behavior. [6] Some 'difficult' children exhibit behavioral problems due to neurological dysfunctions. One study looked specifically at neurological damage and infant behavior in 66 low-birth-weight infants from intact middle-class families. These children exhibited traits such as immaturity, overactivity, temper tantrums, poor attention, and poor school performance. Each of the previous traits listed has been linked to antisocial behavior later in life. [5] However, these children were not followed up with later in life to ensure their trajectory into crime. [5]

Continuity and stability of antisocial behavior

The continuity and stability of antisocial behavior lies at the root of Moffitt's theory. The adolescent limited offenders exhibit antisocial behavior without stability over their lifetime, while life-course-persistent offenders typically display antisocial behavior from very early ages. Biting and hitting as early as age 4 followed by crimes such as shoplifting, selling drugs, theft, robbery, rape, and child abuse characterize a life course persistent offender. [5]

Donker et al. presents a test concerning the prediction on the stability of longitudinal antisocial behavior. Two types of antisocial behavior were measured: covert, or behavior that focuses on deceit and theft, and overt, or behavior that involves direct confrontation and the threat of physical harm. This experiment documents subjects during three main periods of their life: childhood, 6–11 years of age, adolescence, 12–17 years of age, and adulthood, 20–25 years of age. Offenders that begin to show antisocial behavior in childhood that continues into adulthood are what Moffitt considers to be life-course-persistent offenders. Their delinquent behavior is attributed to several factors including neuropsychological impairments and negative environmental features. Moffitt predicts that "…estimates of the individual stability of antisocial behavior are expected to violate the longitudinal law, which states that relationships between variables become weaker as the time interval between them grows longer." [5]

The original sample of children (ages 6–11) in 1983 consisted of 1,125 subjects. Three main areas were studied in the subjects: status violations, overt behavior, and covert behavior. Children exhibiting overt behavior were found to have two times greater risk for covert behavior as an adolescent and three times greater risk for it in adulthood. This violates the longitudinal law and proves Moffitt's expectations correct. Further results also supported this violation, but only with respect to overt behavior, not covert behavior. [7] There is a difference in the continuity of antisocial behavior between men and women as well. In one longitudinal study an entire county's population was followed from age 8 to 48. Only 18% of the women who ranked high in antisocial behavior at age 8 rank high at age 48, while 47% of men stay in the high category. About 37% of both men and women, however, retained low antisocial behavior through age 48. [6]

Life-course-persistent offenders

Biological risk factors

The following biological risk factors have been linked to, but do not cause, persistent antisocial behavior throughout the life course. [3]

Brain Injury

According to multiple studies, a correlation was found between brain insult suffered during delivery and later antisocial behavior. [5] [8] Also in acceleration-deceleration traumatic brain injury (TBI), an increase in aggression and antisocial behavior was recorded after the incident. [9] Perhaps the most well documented injury associated with antisocial behavior is injury to the pre-frontal cortex. Damage to this part of the brain early in childhood correlates to an antisocial behavior that extends through the life-course. [10]

Brain activity

Click to see labeled lobes Four lobes animation small2.gif
Click to see labeled lobes
The right hemisphere is responsible for spatial awareness and orientation. Deficits on this side of the brain can lead to problems in facial and expression recognition. These impairments can do more than just interfere with early attachment and bonding. They can lead to adverse parenting due to the child's inappropriate responses to parental moods or expressions. In one experiment, 868 seven-year-old boys in Pennsylvania were divided into groups: one group on the life-course persistent offender path, one on the adolescent limited path, and one control group. A Continuous Performance Task test (CPT) was used to test frontal lobe function. Larger neurocognitive impairments were found in the life-course persistent group (LCP) than in the control group. Additionally, positron emission tomography (PET), near-infrared spectroscopy, and magnetoencephalography imaging studies have shown more right hemisphere activation during the CPT, so these results are consistent with right hemisphere dysfunction in subjects displaying antisocial behavior. [11]
Reduced glucose metabolism in the pre-frontal cortex has been recorded in murderers compared with normal controls. Also, several other studies cited reduced blood flow in the same area. Recent studies indicate that abnormalities associated with antisocial behavior are localized in the orbitofrontal and dorsolateral prefrontal regions. The dorsolateral prefrontal cortex abnormality may predispose to "response perseveration" leading to a life-course-persistent antisocial behavior despite repeated punishment. [10] Several other abnormalities of the brain have been found with relation to antisocial behavior such as reduced functioning of the amygdala, abnormal glucose metabolism in the temporal lobe, smaller volumes of the hippocampus, and lesser function of the anterior cingulate. [10]
Spatial capabilities
In the previous Pennsylvania study, the life-course persistent (LCP) group showed significant impairments on spatial tests compared with the control group. No significant differences were shown between the adolescent limited (AL) group and the control group with respect to spatial IQ. [11]
Verbal capabilities
Children exhibiting antisocial behavior early in life, many of whom are the same individuals who continue their trajectory into adulthood, often have difficulties with oral communication. [6] [11] Within the 868 Pennsylvania boys, the LCP group had significantly lower verbal IQ (and regular IQ) scores than the adolescent limited group, and no significant differences were shown between the adolescent limited (AL) group and the control group with respect to verbal IQ. [11]

Minor physical anomalies

Moffitt writes, "Minor physical anomalies, which are thought to be observable markers for hidden anomalies in neural development, have been found at elevated rates among violent offenders and subjects with antisocial personality traits." Neural development in the fetus may also be affected by maternal drug abuse, poor prenatal nutrition, or prenatal/postnatal exposure to toxic agents. [5] Minor physical anomalies (MPAs) are features such as low-seated ears, furrowed tongue, and adherent ear lobes. Evidence supporting the link between minor physical anomalies and antisocial behavior shows that the link only exists when adverse environmental factors are present. [8]

Social risk factors

In many studies, the individuals displaying antisocial behavior developed in a family exhibiting "deviant behavior", in an "adverse home environment", or in something similar. [5] However most studies do not specify the exact traits that characterize the tested 'deviant' or 'adverse' environment. Many that are cited include abuse, neglect, socioeconomic status, parental antisocial behavior, etc. There is no evidence that social factors, such as these, can induce antisocial behavior without accompanying biological factors.

Effect of biological and social risk factors together

BiologicalSocial
GeneticAbuse
Brain InjuryNeglect
Brain ActivitySocioeconomic Status
Minor Physical Anomalies (MPAs)Parental deviant behavior

Moffitt projects that initial biological predispositions combined with an adverse rearing environment will initiate the risk of life-course persistent antisocial behavior. She conducted a longitudinal study in New Zealand of boys exhibiting a range of antisocial tendencies. Of the 536 boys, 75 of them had adverse home environments and neuropsychological problems. Those 75 boys scored more than 4 times higher on aggression than the boys with adverse home environments or neuropsychological problems (one of the two). [5] LCP offenders in the Pennsylvania study had higher levels of poverty than the control participants, and they had higher levels of neglect than both the control participants and the AL participants. [11] Additionally, twin studies are often used to isolate effects of nature and nurture. In one such study, the highest criminal activity levels were witnessed in individuals whose foster families exhibited deviant behavior. [5]

Genetic and environmental interactions

The first biological predisposition one thinks of is genetics. Despite Moffitt's original projection that life-course persistent antisocial behavior was more genetically influenced than the adolescent limited variety, a recent study found similar levels of genetic influence on both childhood-onset and adolescent-onset antisocial behavior. [12] Childhood-onset antisocial behavior shares common genetic underpinnings with attention deficit hyperactivity disorder (ADHD) and young adult antisocial behavior, whereas adolescent-onset antisocial behavior does not share any of these common genetic underpinnings. [12] Several experiments use individuals with antisocial parents who have been adopted and raised by other people. Consistently, however, antisocial behavior prevails in the child despite the deviant, biological parents being absent. [3] One such experiment used individuals whose biological parents exhibited criminal behavior and who were adopted. When the adoptive environment was adverse and the genetic predisposition was present, 40% of the adoptees partook in criminal activity compared to only 12.1% with only genetic predispositions. In another study, significant heritability was found for crime, but one subsequent finding was that heritability was higher in individuals from high socioeconomic background and those from rural areas. [8] This shows the link between antisocial behavior and biological risk will be stronger in individuals from kindly social backgrounds than individuals from adverse social backgrounds "because the social causes of crime camouflage the biological contribution. [8] While several studies have been initiated to identify the alleles responsible for antisocial behavior, no such discovery has been made thus far.

Minor physical anomalies and environmental interactions

In one study testing 129 boys from age 12 to 21 years with minor physical anomalies (MPAs), the correlation between antisocial behavior and MPAs only existed when the individual suffering from an MPA was exposed to an adverse home environment. [8] This environment was necessary to express the biological predisposition just like an environment is necessary to express certain genes.

Brain activity and environmental interactions

A link between prefrontal cortex dysfunction and antisocial behavior has been found in many studies. Some frontal lobe lesions have been responsible for impulsivity and disinhibition, which are key characteristics of Antisocial Personality Disorder. [3] In one such study, a group of murderers were divided into two groups: one with benign social backgrounds and one with malignant social backgrounds. Using positron emission tomography (PET) scans were used to detect function in different parts of the brain. Compared to a normal control group, the murderers raised in malignant environments had relatively good prefrontal functioning, but the murderers raised in benign environments had significantly reduced prefrontal functioning, mainly in the right hemisphere. Functional MRI scans were used in another study with violent offenders and abusive environments. Four groups were composed of non-violent controls with no history of abuse, violent offenders with a history of abuse, violent offenders with no history of abuse, and non-violent controls with a history of abuse. The violent offenders who had been abused showed reduced function in the right hemisphere, particularly the right temporal cortex. According to the authors of this article, this experiment's results imply that good right hemispheric functioning may protect against violence in abused children. [8]

Adolescence-limited offenders

Although the biological risk factor do not apply to this group, one point worth noting is that the myelination of the frontal cortex continues into our 20's. [8] This continuing development may help to explain why antisocial behavior ceases after adolescence and why such a spike in crime exists there in the first place.

Cause

According to Terrie Moffitt, there are 3 etiological hypotheses for adolescent-limited offenders:

1. Adolescence-limited antisocial behavior is motivated by the gap between biological maturity and social maturity

2. It is learned from antisocial models who are easily mimicked

3. It is sustained according to the reinforcement principles of learning theory [5]

Neuroethical implications

This type of theory leads to several different neuroethical issues. If, in the future, we were able to use brain scans, behavioral data, or another type of screening to identify life-course-persistent offenders in childhood, what type of interventions would be implemented, if any? Would it even be ethical to use brain scans or other screening methods to preemptively test children in the first place? Assuming that the data was so reliable that there was no chance a child tested to be a life-course-persistent offender could change course throughout his/her life due to social or environmental factors, what would we do with those children? If those positively tested children were placed in a classroom together, away from other children, it is likely that their violence or aggression would simply worsen. Do we want to institute policies that "treat troubled children as future criminals?" [13] One particular experiment compares the neural bases of antisocial behavior and morality. What if, in the future, we could identify the people who had an intact moral compass, but were biologically engineered to exhibit antisocial behavior? Would this change the course of action with these individuals, or does every antisocial individual deserve intervention despite their moral health? [10]

See also

Related Research Articles

<span class="mw-page-title-main">Adolescence</span> Human transition from puberty to adult

Adolescence is a transitional stage of physical and psychological development that generally occurs during the period from puberty to adulthood. Adolescence is usually associated with the teenage years, but its physical, psychological or cultural expressions may begin earlier or end later. Puberty typically begins during preadolescence, particularly in females. Physical growth and cognitive development can extend past the teens. Age provides only a rough marker of adolescence, and scholars have not agreed upon a precise definition. Some definitions start as early as 10 and end as late as 26. The World Health Organization definition officially designates an adolescent as someone between the ages of 10 and 19.

Conduct disorder (CD) is a mental disorder diagnosed in childhood or adolescence that presents itself through a repetitive and persistent pattern of behavior that includes theft, lies, physical violence that may lead to destruction, and reckless breaking of rules, in which the basic rights of others or major age-appropriate norms are violated. These behaviors are often referred to as "antisocial behaviors", and is often seen as the precursor to antisocial personality disorder; however, the latter, by definition, cannot be diagnosed until the individual is 18 years old. Conduct disorder may result from parental rejection and neglect and can be treated with family therapy, as well as behavioral modifications and pharmacotherapy. Conduct disorder is estimated to affect 51.1 million people globally as of 2013.

Antisocial personality disorder is a personality disorder characterized by a limited capacity for empathy and a long-term pattern of disregard or violation of the rights of others. Other notable symptoms include impulsivity and reckless behavior, a lack of remorse after hurting others, deceitfulness, irresponsibility, and aggressive behavior.

<span class="mw-page-title-main">Juvenile delinquency</span> Illegal behavior by minors

Juvenile delinquency, also known as juvenile offending, is the act of participating in unlawful behavior as a minor or individual younger than the statutory age of majority. The term delinquent usually refers to juvenile delinquency, and is also generalised to refer to a young person who behaves an unacceptable way.

Antisocial behaviours are actions which are considered to violate the rights of or otherwise harm others by committing crime or nuisance, such as stealing and physical attack or noncriminal behaviours such as lying and manipulation. It is considered to be disruptive to others in society. This can be carried out in various ways, which includes, but is not limited to, intentional aggression, as well as covert and overt hostility. Anti-social behaviour also develops through social interaction within the family and community. It continuously affects a child's temperament, cognitive ability and their involvement with negative peers, dramatically affecting children's cooperative problem-solving skills. Many people also label behaviour which is deemed contrary to prevailing norms for social conduct as anti-social behaviour. However, researchers have stated that it is a difficult term to define, particularly in the United Kingdom where many acts fall into its category. The term is especially used in British English.

Sex differences in crime are differences between men and women as the perpetrators or victims of crime. Such studies may belong to fields such as criminology, sociobiology, or feminist studies. Despite the difficulty of interpreting them, crime statistics may provide a way to investigate such a relationship from a gender differences perspective. An observable difference in crime rates between men and women might be due to social and cultural factors, crimes going unreported, or to biological factors for example, testosterone or sociobiological theories). The nature of the crime itself may also require consideration as a factor.

Child psychopathology refers to the scientific study of mental disorders in children and adolescents. Oppositional defiant disorder, attention-deficit hyperactivity disorder, and autism spectrum disorder are examples of psychopathology that are typically first diagnosed during childhood. Mental health providers who work with children and adolescents are informed by research in developmental psychology, clinical child psychology, and family systems. Lists of child and adult mental disorders can be found in the International Statistical Classification of Diseases and Related Health Problems, 10th Edition (ICD-10), published by the World Health Organization (WHO) and in the Diagnostic and Statistical Manual of Mental Disorders, Fifth Edition (DSM-5), published by the American Psychiatric Association (APA). In addition, the Diagnostic Classification of Mental Health and Developmental Disorders of Infancy and Early Childhood is used in assessing mental health and developmental disorders in children up to age five.

<span class="mw-page-title-main">Executive functions</span> Cognitive processes necessary for control of behavior

In cognitive science and neuropsychology, executive functions are a set of cognitive processes that are necessary for the cognitive control of behavior: selecting and successfully monitoring behaviors that facilitate the attainment of chosen goals. Executive functions include basic cognitive processes such as attentional control, cognitive inhibition, inhibitory control, working memory, and cognitive flexibility. Higher-order executive functions require the simultaneous use of multiple basic executive functions and include planning and fluid intelligence.

Personality development encompasses the dynamic construction and deconstruction of integrative characteristics that distinguish an individual in terms of interpersonal behavioral traits. Personality development is ever-changing and subject to contextual factors and life-altering experiences. Personality development is also dimensional in description and subjective in nature. That is, personality development can be seen as a continuum varying in degrees of intensity and change. It is subjective in nature because its conceptualization is rooted in social norms of expected behavior, self-expression, and personal growth. The dominant viewpoint in personality psychology indicates that personality emerges early and continues to develop across one's lifespan. Adult personality traits are believed to have a basis in infant temperament, meaning that individual differences in disposition and behavior appear early in life, potentially before language of conscious self-representation develop. The Five Factor Model of personality maps onto the dimensions of childhood temperament. This suggests that individual differences in levels of the corresponding personality traits are present from young ages.

Psychopathy is a mental health condition characterized by persistent antisocial behavior, impaired empathy and remorse, and bold, disinhibited, and egotistical traits. Different conceptions of psychopathy have been used throughout history that are only partly overlapping and may sometimes be contradictory.

Childhood trauma is often described as serious adverse childhood experiences (ACEs). Children may go through a range of experiences that classify as psychological trauma; these might include neglect, abandonment, sexual abuse, emotional abuse, and physical abuse, witnessing abuse of a sibling or parent, or having a mentally ill parent. These events have profound psychological, physiological, and sociological impacts and can have negative, lasting effects on health and well-being such as unsocial behaviors, attention deficit hyperactivity disorder (ADHD), and sleep disturbances. Similarly, children whose mothers have experienced traumatic or stressful events during pregnancy have an increased risk of mental health disorders and other neurodevelopmental disorders.

<span class="mw-page-title-main">Ventromedial prefrontal cortex</span> Body part

The ventromedial prefrontal cortex (vmPFC) is a part of the prefrontal cortex in the mammalian brain. The ventral medial prefrontal is located in the frontal lobe at the bottom of the cerebral hemispheres and is implicated in the processing of risk and fear, as it is critical in the regulation of amygdala activity in humans. It also plays a role in the inhibition of emotional responses, and in the process of decision-making and self-control. It is also involved in the cognitive evaluation of morality.

<span class="mw-page-title-main">Depression in childhood and adolescence</span> Pediatric depressive disorders

Depression is a mental disorder characterized by prolonged unhappiness or irritability. It is accompanied by a constellation of somatic and cognitive signs and symptoms such as fatigue, apathy, sleep problems, loss of appetite, loss of engagement, low self-regard/worthlessness, difficulty concentrating or indecisiveness, or recurrent thoughts of death or suicide.

<span class="mw-page-title-main">Biosocial criminology</span> Psychosocial examination of crime

Biosocial criminology is an interdisciplinary field that aims to explain crime and antisocial behavior by exploring biocultural factors. While contemporary criminology has been dominated by sociological theories, biosocial criminology also recognizes the potential contributions of fields such as behavioral genetics, neuropsychology, and evolutionary psychology.

<span class="mw-page-title-main">Childhood memory</span> Early life experiences often memorable for life

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Terrie Edith Moffitt is an American-British clinical psychologist who is best known for her pioneering research on the development of antisocial behavior and for her collaboration with colleague and partner Avshalom Caspi in research on gene-environment interactions in mental disorders.

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The dual systems model, also known as the maturational imbalance model, is a theory arising from developmental cognitive neuroscience which posits that increased risk-taking during adolescence is a result of a combination of heightened reward sensitivity and immature impulse control. In other words, the appreciation for the benefits arising from the success of an endeavor is heightened, but the appreciation of the risks of failure lags behind.

Adverse childhood experiences (ACEs) include childhood emotional, physical, or sexual abuse and household dysfunction during childhood. The categories are verbal abuse, physical abuse, contact sexual abuse, a battered mother, household substance abuse, household mental illness, incarcerated household members, and parental separation or divorce. The experiences chosen were based upon prior research that has shown to them to have significant negative health or social implications, and for which substantial efforts are being made in the public and private sector to reduce their frequency of occurrence. Scientific evidence is mounting that such adverse childhood experiences (ACEs) have a profound long-term effect on health. Research shows that exposure to abuse and to serious forms of family dysfunction in the childhood family environment are likely to activate the stress response, thus potentially disrupting the developing nervous, immune, and metabolic systems of children. ACEs are associated with lifelong physical and mental health problems that emerge in adolescence and persist into adulthood, including cardiovascular disease, chronic obstructive pulmonary disease, autoimmune diseases, substance abuse, and depression.

References

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