Sanguisorba officinalis var. globularis(Nakai ex T.Kawamoto) W.Lee
Sanguisorba officinalis var. latifoliataLiou & C.Y.Li
Sanguisorba officinalis subsp. microcephala(C.Presl) Calder & Roy L.Taylor
Sanguisorba officinalis f. pallescensAsai
Sanguisorba officinalis f. pilosella(Ohwi) H.Hara
Sanguisorba polygamaF.Nyl.
Sanguisorba praecoxBesser ex Rchb.
Sanguisorba rubraSchrank
Sanguisorba sabaudaMill.
Sanguisorba serotinaJord.
Sanguisorba tauricaJuz.
Sanguisorba unsanensisNakai
The dry fruits contain the achenes which contain the seeds
Sanguisorba officinalis, commonly known as great burnet, is a plant in the family Rosaceae, subfamily Rosoideae. It is native throughout the cooler regions of the Northern Hemisphere in Europe, northern Asia, and northern North America.
Species native to the majority of Europe, Asia and the West Coast of Canada and the United States. It has been introduced to the territories of Finland in Europe and Peru in South America.[4]
Commercial uses
Use is made of its extensive root system for erosion control, as well as a bioremediator, used to reclaim derelict sites such as landfills.
Ornamental
Sanguisorba officinalis is one of several Sanguisorba species cultivated as ornamental plants. The cultivar 'Tanna' is widely available, and has won the Royal Horticultural Society's Award of Garden Merit.[5] The synonym Sanguisorba menziesii is also listed as having gained the Award.[6]
Ethnomedical uses
It has been used in traditional Chinese medicine (TCM) where it is known by the name Di Yu. It is said to cool the blood, stop bleeding, clear heat, and heal wounds (Chinese Herbal Materia Medica by Dan Bensky).
↑ Bastow KF, Bori ID, Fukushima Y, Kashiwada Y, Tanaka T, Nonaka G, Nishioka I, Lee KH (June 1993). "Inhibition of DNA topoisomerases by sanguiin H-6, a cytotoxic dimeric ellagitannin from Sanguisorba officinalis". Planta Med. 59 (3): 240–5. doi:10.1055/s-2006-959659. PMID8391144.
↑ Zhu X, Wang K, Zhang K, Zhu L, Zhou F (May 2014). "Ziyuglycoside II induces cell cycle arrest and apoptosis through activation of ROS/JNK pathway in human breast cancer cells". Toxicol. Lett. 227 (1): 65–73. doi:10.1016/j.toxlet.2014.03.015. PMID24680927.
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