Right bundle branch block

Last updated
Right bundle branch block
Right bundle branch block (RBBB) of the human heart.svg
An illustration of a right bundle branch block located in intraventricular septum
Right bundle branch block ECG characteristics.svg
ECG characteristics of a typical RBBB showing wide QRS complexes with a terminal R wave in lead V1 and a prolonged S wave in lead V6.
Specialty Cardiology
Typescomplete right bundle branch block (CRBBB)
incomplete right bundle branch block (IRBBB)

A right bundle branch block (RBBB) is a heart block in the right bundle branch of the electrical conduction system. [1]

Contents

During a right bundle branch block, the right ventricle is not directly activated by impulses travelling through the right bundle branch. The left ventricle, however, is still normally activated by the left bundle branch. These impulses are then able to travel through the myocardium of the left ventricle to the right ventricle and depolarize the right ventricle this way. As conduction through the myocardium is slower than conduction through the bundle of His-Purkinje fibres, the QRS complex is seen to be widened. The QRS complex often shows an extra deflection that reflects the rapid depolarisation of the left ventricle followed by the slower depolarisation of the right ventricle.

Incomplete right bundle branch block

Incomplete right bundle branch block (IRBBB) is a conduction abnormality in the right bundle branch block. While a complete RBBB has a QRS duration of 120 ms or more, an incomplete block has a wave duration between 100 and 120 ms. It has a relatively high prevalence, a study conducted on young Swiss military conscripts with a mean age of 19 years found a prevalence of 13.5%. [2] It affects patients of all ages, more commonly males and athletes, however it is not always a benign finding. Therefore, if abnormalities are detected on the physical exam, further testing should be done to exclude heart disease. [3]

There is no consensus in the literature regarding criteria for diagnosis. However, according to the American Heart Association/American College of Cardiology Foundation/Heart Rhythm Foundation (AHA/ACCF/HRS) it is defined by the following finding in adults:

  1. QRS wave duration between 100 and 120 ms.
  2. rsr, rsR, or rSR in leads V1 or V2.
  3. S wave of longer duration than R wave or greater than 40 ms in leads I and V6.
  4. Normal R wave peak time in both V5 and V6, but greater than 50 ms in V1.

The first three criteria are needed for diagnosis. The fourth is needed when a pure dominant R waver is present on V1. [3]

Causes

Common causes include normal variation, changes in bundle branch structure - such as mechanical stretching, chest trauma, right ventricular hypertrophy or strain, congenital heart disease such as atrial septal defect, and ischemic heart disease. [4] In addition, a right bundle branch block may also result from Brugada syndrome, Chagas disease, [5] [6] pulmonary embolism, rheumatic heart disease, myocarditis, cardiomyopathy, or hypertension. [7]

Causes for incomplete right bundle branch block (IRBBB) often involve exercise-induced right ventricular remodeling, increased right ventricular (RV) free wall thickness, especially in athletes due to prolonged endurance exercise. [8]

Diagnosis

Reizleitungssystem 12.png
Normal electrical conduction system of the heart (Schematic). All myocardial segments are excited almost simultaneously (purple staining).
Reizleitungssystem RSB.png
Conduction in RBBB (Schematic): With a blockage in the right bundle branch (red), the left ventricle is excited in time (purple), while the excitation of the right ventricle takes a detour via the left bundle branch (blue arrows).

The criteria to diagnose a right bundle branch block on the electrocardiogram:

The T wave should be deflected opposite the terminal deflection of the QRS complex. This is known as appropriate T wave discordance with bundle branch block. A concordant T wave may suggest ischemia or myocardial infarction.[ citation needed ]

Treatment

The underlying condition may be treated by medications to control hypertension or diabetes, if they are the primary underlying cause. If coronary arteries are blocked, an invasive coronary angioplasty may relieve the impending RBBB. [10]

Epidemiology

Prevalence of RBBB increases with age due to changes in the heart's conduction system. It's estimated up to 11.3% of the population by the age of 80 have RBBB. [11]

See also

Related Research Articles

<span class="mw-page-title-main">Bradycardia</span> Heart rate below the normal range

Bradycardia, also called bradyarrhythmia, is a resting heart rate under 60 beats per minute (BPM). While bradycardia can result from various pathologic processes, it is commonly a physiologic response to cardiovascular conditioning or due to asymptomatic type 1 atrioventricular block. Resting heart rates of less than 50 BPM are often normal during sleep in young and healthy adults and athletes. In large population studies of adults without underlying heart disease, resting heart rates of 45-50 BPM appear to be the lower limits of normal, dependent on age and sex. Bradycardia is most likely to be discovered in the elderly, as age and underlying cardiac disease progression contribute to its development.

<span class="mw-page-title-main">Electrocardiography</span> Examination of the hearts electrical activity

Electrocardiography is the process of producing an electrocardiogram, a recording of the heart's electrical activity through repeated cardiac cycles. It is an electrogram of the heart which is a graph of voltage versus time of the electrical activity of the heart using electrodes placed on the skin. These electrodes detect the small electrical changes that are a consequence of cardiac muscle depolarization followed by repolarization during each cardiac cycle (heartbeat). Changes in the normal ECG pattern occur in numerous cardiac abnormalities, including:

<span class="mw-page-title-main">Ventricular fibrillation</span> Rapid quivering of the ventricles of the heart

Ventricular fibrillation is an abnormal heart rhythm in which the ventricles of the heart quiver. It is due to disorganized electrical activity. Ventricular fibrillation results in cardiac arrest with loss of consciousness and no pulse. This is followed by sudden cardiac death in the absence of treatment. Ventricular fibrillation is initially found in about 10% of people with cardiac arrest.

<span class="mw-page-title-main">Wolff–Parkinson–White syndrome</span> Medical condition

Wolff–Parkinson–White syndrome (WPWS) is a disorder due to a specific type of problem with the electrical system of the heart involving an accessory pathway able to conduct electrical current between the atria and the ventricles, thus bypassing the atrioventricular node. About 60% of people with the electrical problem developed symptoms, which may include an abnormally fast heartbeat, palpitations, shortness of breath, lightheadedness, or syncope. Rarely, cardiac arrest may occur. The most common type of irregular heartbeat that occurs is known as paroxysmal supraventricular tachycardia.

<span class="mw-page-title-main">Arrhythmogenic cardiomyopathy</span> Medical condition

Arrhythmogenic cardiomyopathy (ACM), arrhythmogenic right ventricular dysplasia (ARVD), or arrhythmogenic right ventricular cardiomyopathy (ARVC), most commonly is an inherited heart disease.

<span class="mw-page-title-main">Cardiac conduction system</span> Aspect of heart function

The cardiac conduction system transmits the signals generated by the sinoatrial node – the heart's pacemaker, to cause the heart muscle to contract, and pump blood through the body's circulatory system. The pacemaking signal travels through the right atrium to the atrioventricular node, along the bundle of His, and through the bundle branches to Purkinje fibers in the walls of the ventricles. The Purkinje fibers transmit the signals more rapidly to stimulate contraction of the ventricles.

<span class="mw-page-title-main">Ventricular tachycardia</span> Medical condition of the heart

Ventricular tachycardia is a cardiovascular disorder in which fast heart rate occurs in the ventricles of the heart. Although a few seconds of VT may not result in permanent problems, longer periods are dangerous; and multiple episodes over a short period of time are referred to as an electrical storm. Short periods may occur without symptoms, or present with lightheadedness, palpitations, shortness of breath, chest pain, and decreased level of consciousness. Ventricular tachycardia may lead to coma and persistent vegetative state due to lack of blood and oxygen to the brain. Ventricular tachycardia may result in ventricular fibrillation (VF) and turn into cardiac arrest. This conversion of the VT into VF is called the degeneration of the VT. It is found initially in about 7% of people in cardiac arrest.

<span class="mw-page-title-main">Ebstein's anomaly</span> Congenital heart defect

Ebstein's anomaly is a congenital heart defect in which the septal and posterior leaflets of the tricuspid valve are displaced downwards towards the apex of the right ventricle of the heart. EA has great anatomical heterogeneity that generates a wide spectrum of clinical features at presentation and is complicated by the fact that the lesion is often accompanied by other congenital cardiac lesions. It is classified as a critical congenital heart defect accounting for less than 1% of all congenital heart defects presenting in around 1 per 200,000 live births. Ebstein's anomaly usually presents with a systolic murmur and frequently with a gallop rhythm.

<span class="mw-page-title-main">Left ventricular hypertrophy</span> Medical condition

Left ventricular hypertrophy (LVH) is thickening of the heart muscle of the left ventricle of the heart, that is, left-sided ventricular hypertrophy and resulting increased left ventricular mass.

<span class="mw-page-title-main">T wave</span> Repolarization of the ventricles in a human heart

In electrocardiography, the T wave represents the repolarization of the ventricles. The interval from the beginning of the QRS complex to the apex of the T wave is referred to as the absolute refractory period. The last half of the T wave is referred to as the relative refractory period or vulnerable period. The T wave contains more information than the QT interval. The T wave can be described by its symmetry, skewness, slope of ascending and descending limbs, amplitude and subintervals like the Tpeak–Tend interval.

<span class="mw-page-title-main">Bundle branch block</span> Medical condition

A bundle branch block is a partial or complete interruption in the flow of electrical impulses in either of the bundle branches of the heart's electrical system.

<span class="mw-page-title-main">QRS complex</span> Represents ventricular depolarization, which results in ventricular contraction

The QRS complex is the combination of three of the graphical deflections seen on a typical electrocardiogram. It is usually the central and most visually obvious part of the tracing. It corresponds to the depolarization of the right and left ventricles of the heart and contraction of the large ventricular muscles.

<span class="mw-page-title-main">Atrioventricular block</span> Medical condition

Atrioventricular block is a type of heart block that occurs when the electrical signal traveling from the atria, or the upper chambers of the heart, to ventricles, or the lower chambers of the heart, is impaired. Normally, the sinoatrial node produces an electrical signal to control the heart rate. The signal travels from the SA node to the ventricles through the atrioventricular node. In an AV block, this electrical signal is either delayed or completely blocked. When the signal is completely blocked, the ventricles produce their own electrical signal to control the heart rate. The heart rate produced by the ventricles is much slower than that produced by the SA node.

<span class="mw-page-title-main">Left bundle branch block</span> Medical condition

Left bundle branch block (LBBB) is a conduction abnormality in the heart that can be seen on an electrocardiogram (ECG). In this condition, activation of the left ventricle of the heart is delayed, which causes the left ventricle to contract later than the right ventricle.

Lown–Ganong–Levine syndrome (LGL) is a pre-excitation syndrome of the heart. Those with LGL syndrome have episodes of abnormal heart racing with a short PR interval and normal QRS complexes seen on their electrocardiogram when in a normal sinus rhythm. LGL syndrome was originally thought to be due to an abnormal electrical connection between the atria and the ventricles, but is now thought to be due to accelerated conduction through the atrioventricular node in the majority of cases. The syndrome is named after Bernard Lown, William Francis Ganong, Jr., and Samuel A. Levine.

An accessory pathway is an additional electrical connection between two parts of the heart. These pathways can lead to abnormal heart rhythms or arrhythmias associated with symptoms of palpitations. Some pathways may activate a region of ventricular muscle earlier than would normally occur, referred to as pre-excitation, and this may be seen on an electrocardiogram. The combination of an accessory pathway that causes pre-excitation with arrhythmias is known as Wolff-Parkinson-White syndrome.

<span class="mw-page-title-main">Right axis deviation</span> Medical condition

The electrical axis of the heart is the net direction in which the wave of depolarization travels. It is measured using an electrocardiogram (ECG). Normally, this begins at the sinoatrial node ; from here the wave of depolarisation travels down to the apex of the heart. The hexaxial reference system can be used to visualise the directions in which the depolarisation wave may travel.

A left posterior fascicular block (LPFB), also known as left posterior hemiblock (LPH), is a condition where the left posterior fascicle, which travels to the inferior and posterior portion of the left ventricle, does not conduct the electrical impulses from the atrioventricular node. The wave-front instead moves more quickly through the left anterior fascicle and right bundle branch, leading to a right axis deviation seen on the ECG.

<span class="mw-page-title-main">Intraventricular block</span> Medical condition

An intraventricular block is a heart conduction disorder — heart block of the ventricles of the heart. An example is a right bundle branch block, right fascicular block, bifascicular block, trifascicular block.

<span class="mw-page-title-main">Left axis deviation</span> Heart condition

In electrocardiography, left axis deviation (LAD) is a condition wherein the mean electrical axis of ventricular contraction of the heart lies in a frontal plane direction between −30° and −90°. This is reflected by a QRS complex positive in lead I and negative in leads aVF and II.

References

  1. "Conduction Blocks". Department of Physiology. Kansas City University of Medicine & Biosciences. 2006. Archived from the original on 9 May 2009. Retrieved 20 January 2009.
  2. Kobza R, Cuculi F, Abächerli R, Toggweiler S, Suter Y, Frey F, et al. (December 2012). "Twelve-lead electrocardiography in the young: physiologic and pathologic abnormalities". Heart Rhythm. 9 (12): 2018–2022. doi:10.1016/j.hrthm.2012.08.034. PMID   23102624.
  3. 1 2 Floria M, Parteni N, Neagu AI, Sascau RA, Statescu C, Tanase DM (June 2021). "Incomplete right bundle branch block: Challenges in electrocardiogram diagnosis". Anatolian Journal of Cardiology. 25 (6): 380–384. doi:10.5152/AnatolJCardiol.2021.84375. PMC   8210929 . PMID   34100724. S2CID   235368614.
  4. Goldman L (2011). Goldman's Cecil Medicine (24th ed.). Philadelphia: Elsevier Saunders. pp.  400–401. ISBN   978-1-4377-2788-3.
  5. "Chagas Disease: What U.S. Clinicians Need to Know". Centers for Disease Control and Prevention. CDC. Retrieved 3 October 2023.
  6. Rojas LZ, Glisic M, Pletsch-Borba L, Echeverría LE, Bramer WM, Bano A, et al. (June 2018). "Electrocardiographic abnormalities in Chagas disease in the general population: A systematic review and meta-analysis". PLoS Neglected Tropical Diseases. 12 (6): e0006567. doi: 10.1371/journal.pntd.0006567 . PMC   5999094 . PMID   29897909.
  7. Ikeda T (January 2021). "Right Bundle Branch Block: Current Considerations". Current Cardiology Reviews. 17 (1): 24–30. doi:10.2174/1573403X16666200708111553. PMC   8142372 . PMID   32640959.
  8. Floria M, Parteni N, Neagu AI, Sascau RA, Statescu C, Tanase DM (June 2021). "Incomplete right bundle branch block: Challenges in electrocardiogram diagnosis". Anatolian Journal of Cardiology. 25 (6): 380–384. doi:10.5152/AnatolJCardiol.2021.84375. PMC   8210929 . PMID   34100724. S2CID   235368614.
  9. Yanowitz FG. "Lesson VI - ECG Conduction Abnormalities". University of Utah School of Medicine. Archived from the original on 26 January 2008. Retrieved 2009-01-07.
  10. "Right Bundle Branch Block". Symptoma. Retrieved 2015-08-13.
  11. Harkness WT, Hicks M (2022). "Right Bundle Branch Block". StatPearls. Treasure Island (FL): StatPearls Publishing. PMID   29939649 . Retrieved 2022-02-24.